No abstract available
Keywords:
biomarkers; cancer metabolism; immune evasion; mitochondrial dysfunction; mitochondrial-targeted therapies; personalized therapy; programmed cell death; tumor microenvironment.
Grants and funding
The authors declare that financial support was received for the research, authorship, and/or publication of this article. This study is supported by the National Natural Science Foundation of China (81802504) and the Sichuan Science and Technology Program (2023YFH0010).