Exopolysaccharides from Bifidobacterium longum subsp. infantis and Bifidobacterium adolescentis modulate Toll-like receptor signaling

Carbohydr Polym. 2025 Feb 1;349(Pt B):123017. doi: 10.1016/j.carbpol.2024.123017. Epub 2024 Nov 15.

Abstract

Background: Exopolysaccharides (EPS) from probiotic bacteria like bifidobacteria, have gained considerable attention for the beneficial effects they exert in the gastrointestinal environment. Here, we investigated whether EPS isolated from Bifidobacterium longum subsp. infantis and Bifidobacterium adolescentis can interact with Toll-like receptors (TLRs) in a structure-dependent way and subsequently we investigated whether they influence cytokine-production in dendritic cells (DCs).

Results: EPS from both B. infantis and B. adolescentis were found to be structurally different and were able to inhibit signaling of TLR2 and TLR4 in an EPS-type dependent fashion. EPS from B. infantis was shown to have stronger inhibitory effects on TLR2/1, whereas EPS from B. adolescentis showed stronger effects for TLR2/6 and TLR4. Incubation of DCs with EPS alone had no effect, however stimulation of DCs with spend-medium of epithelial cells incubated with EPS reduced production of the cytokines MCP-1/CCL2 and TNFα.

Conclusion: Here we show that EPS from B. infantis and B. adolescentis have structure-dependent immunomodulatory effects, indicating that EPS might be important effector molecules responsible for the health benefits of bifidobacteria.

Keywords: Bifidobacteria; Dendritic cells; Exopolysaccharides; Immunomodulation; Toll-like receptors.

MeSH terms

  • Bifidobacterium adolescentis* / metabolism
  • Bifidobacterium longum subspecies infantis / metabolism
  • Bifidobacterium* / metabolism
  • Dendritic Cells* / drug effects
  • Dendritic Cells* / metabolism
  • Humans
  • Polysaccharides, Bacterial* / chemistry
  • Polysaccharides, Bacterial* / pharmacology
  • Probiotics / pharmacology
  • Signal Transduction* / drug effects
  • Toll-Like Receptor 2 / metabolism
  • Toll-Like Receptor 4 / metabolism
  • Toll-Like Receptors / metabolism

Substances

  • Polysaccharides, Bacterial
  • Toll-Like Receptor 2
  • Toll-Like Receptor 4
  • Toll-Like Receptors