Inhibition of platelet function with 2,3-dihydroxybenzoic acid

Br J Haematol. 1977 Dec;37(4):569-77. doi: 10.1111/j.1365-2141.1977.tb01030.x.

Abstract

2,3-Dihydroxybenzoic acid (2,3-DHB) inhibits the second wave of platelet aggregation release of serotonin, and malonaldehyde production. The effect is concentration dependent and reversible in vitro and in vivo. 50% inhibition of platelet aggregation induced in vitro with 70 micron adrenaline was obtained with 1.5 mM 2,3-DHB. Malonaldehyde production was inhibited by 50% with 0.4 mM 2,3-DHB. Serotonin release was 50% inhibited by 50% with 0.08 mm 2,3-DHB. Neither desferrioxamine nor various benzoic acid derivatives with iron-chelating properties showed the effect. Only benzoic acid derivatives with the potential to terminate free radical reactions through the formation of an ortho- or paraquinone structure inhibit the second wave of platelet aggregation. These studies support the concept that the second wave of platelet aggregation is mediated through free radical reactions.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Blood Platelets / drug effects*
  • Blood Platelets / metabolism
  • Depression, Chemical
  • Humans
  • Hydroxybenzoates / pharmacology*
  • In Vitro Techniques
  • Malondialdehyde / metabolism
  • Platelet Aggregation / drug effects
  • Secretory Rate / drug effects
  • Serotonin / metabolism

Substances

  • Hydroxybenzoates
  • Serotonin
  • Malondialdehyde