Corticotropin-releasing factor stimulates Ca2+ influx in cultured rat astrocytes

Biochem Biophys Res Commun. 1994 Mar 30;199(3):1103-7. doi: 10.1006/bbrc.1994.1344.

Abstract

Corticotropin-releasing factor (CRF) increased intracellular Ca2+ concentration in single astrocytes. The effect in increasing intracellular Ca2+ was not observed in Ca2(+)-free solution. Furthermore, CRF at concentrations more than 10 nM stimulated 45Ca2+ uptake in cultured rat astrocytes. The action was blocked by alpha-helical CRF(9-41) in a competitive manner, but not by nifedipine and 3,4-dichlorobenzamil. On the other hand, CRF did not stimulate cAMP formation, cGMP formation and phosphoinositide hydrolysis in astrocytes. These results indicate that CRF increases Ca2+ influx via an activation of CRF receptors in a cAMP-independent mechanism in cultured astrocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 1-Methyl-3-isobutylxanthine / pharmacology
  • Amiloride / pharmacology
  • Animals
  • Animals, Newborn
  • Astrocytes / drug effects
  • Astrocytes / metabolism*
  • Calcium / metabolism*
  • Cells, Cultured
  • Cerebral Cortex / metabolism*
  • Corticotropin-Releasing Hormone / pharmacology*
  • Cyclic AMP / metabolism
  • Cyclic GMP / metabolism
  • Kinetics
  • Nifedipine / pharmacology
  • Phosphatidylinositols / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Time Factors

Substances

  • Phosphatidylinositols
  • Amiloride
  • Corticotropin-Releasing Hormone
  • Cyclic AMP
  • Cyclic GMP
  • Nifedipine
  • Calcium
  • 1-Methyl-3-isobutylxanthine