Glutamate receptors induce a burst of superoxide via activation of nitric oxide synthase in arginine-depleted neurons

J Biol Chem. 1994 Apr 29;269(17):12589-93.

Abstract

We have previously shown in cultured cerebellar granule neurons (Lafon-Cazal, M., Pietri, S., Culcasi, M., and Bockaert, J. (1993) Nature 364, 535-537) that upon N-methyl-D-aspartate stimulation, a nitric oxide synthase (NOS)-independent, arachidonic acid-dependent generation of superoxide free radicals (O2-.) is observed after a lag time of 10-15 min. Using the electron spin resonance spin trapping technique, we show that N-methyl-D-aspartate stimulation produced a more rapid burst of O2-. in L-arginine (L-Arg)-depleted neurons. These O2-. radicals are synthesized by NOS. KCl and kainate, which also stimulated NOS in these neurons, produced this rapid burst of O2-., which was blocked as follows: (a) in the presence of L-NG-nitro-arginine (L-Narg), and (b) by L-Arg repletion. This burst of O2-. was arachidonic acid-independent, and its time course was similar to that of nitric oxide production. It was also responsible for a weak but significant cell death that was suppressed by L-Narg and L-Arg.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Oxidoreductases / metabolism*
  • Animals
  • Arginase / metabolism
  • Arginine / deficiency
  • Arginine / metabolism*
  • Brain / cytology
  • Brain / drug effects
  • Brain / metabolism
  • Cell Death
  • Cells, Cultured
  • Cyclic GMP / metabolism
  • Electron Spin Resonance Spectroscopy
  • Enzyme Activation
  • Male
  • Mice
  • N-Methylaspartate / pharmacology
  • Neurons / metabolism*
  • Nitric Oxide Synthase
  • Receptors, Glutamate / physiology*
  • Superoxides / metabolism*

Substances

  • Receptors, Glutamate
  • Superoxides
  • N-Methylaspartate
  • Arginine
  • Nitric Oxide Synthase
  • Amino Acid Oxidoreductases
  • Arginase
  • Cyclic GMP