Blockade of neurotensin receptors by the antagonist SR 48692 partially prevents retrograde axonal transport of neurotensin in rat nigrostriatal system

Neurosci Lett. 1994 Jan 17;166(1):106-8. doi: 10.1016/0304-3940(94)90851-6.

Abstract

The effect of SR 48692, a potent and selective non-peptide antagonist of the neurotensin receptor, was investigated on the retrograde axonal transport of neurotensin in the rat nigrostriatal dopamine pathway. When rats were injected in the striatum with (3-[125I]iodotyrosyl3)neurotensin, a substantial accumulation of radioactivity appeared in the ipsilateral substantia nigra 1.5 h after injection, and highest levels (336 +/- 23 dpm/mg of protein) were observed 2.5-3.5 h after the injection. The phenomenon required a pretreatment of the animals with thiorphan (30 micrograms) an inhibitor of endopeptidase. The amount of radioactivity accumulated (3.5 h) was found to be reduced (25%) by local (100 nM) or peripheral administration of SR 48692 (5, 10, 20 mg/kg, i.p.; 25%, 40%, 40%, respectively). Our results indicate that blockade of neurotensin receptors by a selective non-peptide receptor antagonist affects the retrograde axonal transport of the tridecapeptide, and further suggest the notion that this process involves neurotensin receptors.

MeSH terms

  • Animals
  • Axonal Transport / drug effects*
  • Dopamine / metabolism
  • Iodine Radioisotopes
  • Male
  • Neostriatum / drug effects
  • Neostriatum / metabolism*
  • Neurotensin / metabolism*
  • Pyrazoles / pharmacology*
  • Quinolines / pharmacology*
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, Neurotensin / antagonists & inhibitors*
  • Substantia Nigra / drug effects
  • Substantia Nigra / metabolism*

Substances

  • Iodine Radioisotopes
  • Pyrazoles
  • Quinolines
  • Receptors, Neurotensin
  • SR 48527
  • SR 48692
  • Neurotensin
  • Dopamine