Inhibitory effect of cyclosporin A and FK506 on nitric oxide production by cultured macrophages. Evidence of a direct effect on nitric oxide synthase activity

Immunology. 1995 Mar;84(3):476-81.

Abstract

Casein-elicited peritoneal macrophages from mice were cultured either alone or with interferon-gamma (IFN-gamma) and bacterial lipopolysaccharide (LPS), and the effect of cyclosporin A (CsA) and FK506 on NO2- production (due technical difficulties NO2- was taken as the index for NO) was analysed. We observed an inhibitory effect of CsA and FK506 on NO2- production. The IC50 for NO2- production by casein-elicited macrophages was 0.1 microgram/ml for CsA and 0.3 microgram/ml FK506. The effect of both drugs was dose-dependent and was more clear in non-stimulated macrophages. The presence of IFN-gamma and LPS in the culture increased NO2- production by casein-elicited macrophages and partially eliminated the inhibition exerted by CsA and FK506. Both drugs acted directly on the nitric oxide synthase (NOS), since CsA and FK506 reduced by 35% and by 17%, respectively, NOS activity in the crude cytosolic fraction. However, CsA and FK506 did not alter 14CO2 production from [1-14C]glucose, suggesting that the pentose monophosphate pathway activity was not modified. These data add new insight into the interpretation of the immunosuppressive properties of both drugs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Oxidoreductases / drug effects*
  • Amino Acid Oxidoreductases / metabolism
  • Animals
  • Cells, Cultured
  • Cyclosporine / pharmacology*
  • Dose-Response Relationship, Drug
  • Kinetics
  • Lactates / biosynthesis
  • Lactic Acid
  • Macrophages, Peritoneal / drug effects*
  • Macrophages, Peritoneal / metabolism
  • Male
  • Mice
  • Mice, Inbred Strains
  • Nitric Oxide / biosynthesis*
  • Nitric Oxide Synthase
  • Pentose Phosphate Pathway / drug effects
  • Tacrolimus / pharmacology*

Substances

  • Lactates
  • Nitric Oxide
  • Lactic Acid
  • Cyclosporine
  • Nitric Oxide Synthase
  • Amino Acid Oxidoreductases
  • Tacrolimus