The kinetics of allergen-induced transcription of messenger RNA for monocyte chemotactic protein-3 and RANTES in the skin of human atopic subjects: relationship to eosinophil, T cell, and macrophage recruitment

J Exp Med. 1995 Jun 1;181(6):2153-9. doi: 10.1084/jem.181.6.2153.

Abstract

The C-C chemokines RANTES and monocyte chemotactic protein-3 (MCP-3) are potent chemoattractants in vitro for eosinophils and other cell types associated with allergic reactions. We tested the hypothesis that the allergen-induced infiltration of eosinophils, T cells, and macrophages in the skin of atopic subjects is accompanied by the appearance of mRNA+ cells for RANTES and MCP-3. Cryostat sections were obtained from skin biopsies from six subjects 6, 24, and 48 h after allergen challenge. Tissue was processed for immunocytochemistry (ICC) and for in situ hybridization using 35S-labeled riboprobes for RANTES and MCP-3. In contrast to diluent controls, allergen provoked a significant increase in mRNA+ cells for MCP-3, which peaked at 6 h and progressively declined at 24 and 48 h. This paralleled the kinetics of total (major basic protein positive [MBP]+) and activated (cleaved form of eosinophil cationic protein [EG2]+) eosinophil infiltration. The allergen-induced expression of cells mRNA+ for RANTES was also clearly demonstrable at 6 h. However, the numbers were maximal at 24 h and declined slightly at the 48-h time point. The number of mRNA+ cells for RANTES paralleled the kinetics of infiltration of CD3+, CD4+, and CD8+ T cells whereas the number of CD68+ macrophages was still increasing at 48 h. These data support the view that MCP-3 is involved in the regulation of the early eosinophil response to specific allergen, whereas RANTES may have more relevance to the later accumulation of T cells and macrophages.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Allergens*
  • Biopsy
  • Chemokine CCL5
  • Chemokine CCL7
  • Chemotactic Factors / biosynthesis*
  • Cytokines*
  • Dermatitis, Atopic / immunology*
  • Dermatitis, Atopic / metabolism
  • Dermatitis, Atopic / pathology
  • Eosinophils / immunology*
  • Female
  • Gene Expression*
  • Humans
  • Kinetics
  • Lymphokines / biosynthesis*
  • Macrophages / immunology*
  • Male
  • Monocyte Chemoattractant Proteins*
  • RNA, Messenger / biosynthesis
  • Skin / immunology*
  • Skin / metabolism
  • Skin / pathology
  • T-Lymphocytes / immunology*
  • Transcription, Genetic*

Substances

  • Allergens
  • CCL7 protein, human
  • Chemokine CCL5
  • Chemokine CCL7
  • Chemotactic Factors
  • Cytokines
  • Lymphokines
  • Monocyte Chemoattractant Proteins
  • RNA, Messenger