Abstract
Objective:
To determine the role of opioidergic and dopaminergic activity in the suppression of GnRH0LH in a hyperprolactinemic state.
Design:
Case report.
Setting:
University hospital.
Patient:
A 68-year-old woman with a macroprolactinoma.
Interventions:
Serial 10-hour IV infusions of naloxone and metoclopramide.
Main outcome measure:
Serum LH concentration.
Results:
Naloxone induced a small but significant rise of serum LH levels, which displayed a pulsatile pattern. By contrast, metoclopramide elicited no significant response in LH secretion.
Conclusion:
Opioidergic but not dopaminergic neurotransmission plays a direct role in the suppression of LH secondary to hyperprolactinemia.
MeSH terms
-
Aged
-
Bromocriptine / pharmacology
-
Dopamine Agonists / pharmacology
-
Dopamine Antagonists / administration & dosage
-
Dopamine Antagonists / pharmacology*
-
Female
-
Follicle Stimulating Hormone / blood
-
Humans
-
Hyperprolactinemia / blood*
-
Hypogonadism / blood*
-
Injections, Intravenous
-
Luteinizing Hormone / blood*
-
Metoclopramide / administration & dosage
-
Metoclopramide / pharmacology*
-
Naloxone / administration & dosage
-
Naloxone / pharmacology*
-
Narcotic Antagonists / administration & dosage
-
Narcotic Antagonists / pharmacology*
-
Pituitary Neoplasms / blood
-
Postmenopause / blood*
-
Prolactin / blood
-
Prolactinoma / blood
Substances
-
Dopamine Agonists
-
Dopamine Antagonists
-
Narcotic Antagonists
-
Naloxone
-
Bromocriptine
-
Prolactin
-
Luteinizing Hormone
-
Follicle Stimulating Hormone
-
Metoclopramide