Tumor selective effect of RS-1541 (palmitoyl-rhizoxin) in M5076 sarcoma and host tissues in vivo

Pharm Res. 1995 Mar;12(3):370-5. doi: 10.1023/a:1016248417492.

Abstract

RS-1541 is a 13-O-palmitoyl derivative of rhizoxin, an inhibitor of tubulin polymerization. After intravenous administration of RS-1541 to mice bearing M5076 sarcoma, the maximal inhibitory effect of RS-1541 on DNA synthesis in the tumor was observed 24 h after administration, in agreement with the Cmax of rhizoxin produced from RS-1541, but not with the Cmax of RS-1541. The inhibitory effect after RS-1541 was much higher than that after rhizoxin itself. In the spleen, thymus and bone marrow, DNA synthesis was strongly inhibited by rhizoxin but not by RS-1541. After administration of RS-1541, no significant amounts of rhizoxin were detected in the tissues, except for the tumor. In acute toxicity tests, RS-1541 appeared to be less toxic than rhizoxin. These results indicate that RS-1541 possesses a high tumor-selective effect compared with rhizoxin, because of the selective production of rhizoxin in the tumor after administration of RS-1541.

MeSH terms

  • Animals
  • Antibiotics, Antineoplastic / pharmacology*
  • Antibiotics, Antineoplastic / toxicity
  • DNA, Neoplasm / biosynthesis*
  • Dose-Response Relationship, Drug
  • Female
  • Injections, Intravenous
  • Lactones / pharmacology
  • Lactones / toxicity
  • Macrolides
  • Male
  • Mice
  • Sarcoma, Experimental / metabolism
  • Thymidine / metabolism*
  • Time Factors

Substances

  • Antibiotics, Antineoplastic
  • DNA, Neoplasm
  • Lactones
  • Macrolides
  • palmitoyl rhizoxin
  • rhizoxin
  • Thymidine