Regulation of cytosolic calcium by collagen in single human platelets

Br J Pharmacol. 1995 May;115(1):101-6. doi: 10.1111/j.1476-5381.1995.tb16326.x.

Abstract

1. There is controversy in the literature as to whether collagen is able to induce directly a rise in cytosolic calcium concentration ([Ca2+]i) in human platelets. We have addressed this question by observing the cytosolic calcium response of single fura-2-loaded human platelets settling onto a collagen-coated surface using dynamic fluorescence ratio imaging. 2. Following a short lag phase after adherence to collagen fibres, platelets underwent a rapid rise in cytosolic calcium from basal values of 80 +/- 13 nM (n = 24) to a peak of 475 +/- 42 nM (n = 24) which was sustained for the remaining period of the experiment. 3. The tyrphostin protein tyrosine kinase inhibitor, ST271, reduced substantially the proportion of platelets which exhibited a rise in [Ca2+]i on adherence to collagen and transformed the response in remaining cells to one of oscillations. 4. In contrast, and as a control for collagen, laminin-coated surfaces induced adherence of human platelets without elevating intracellular [Ca2+]; the cells however remained responsive to ADP. 5. We conclude that collagen directly induces a rise in cytosolic calcium in single human platelets through a tyrosine kinase-mediated pathway.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Diphosphate / pharmacology
  • Blood Platelets / drug effects
  • Blood Platelets / metabolism*
  • Calcium / metabolism*
  • Cell Adhesion / physiology
  • Collagen / pharmacology*
  • Cytosol / metabolism
  • Humans
  • In Vitro Techniques
  • Inositol Phosphates / biosynthesis
  • Laminin / pharmacology
  • Protein-Tyrosine Kinases / antagonists & inhibitors
  • Styrenes / pharmacology

Substances

  • Inositol Phosphates
  • Laminin
  • Styrenes
  • ST 271
  • Adenosine Diphosphate
  • Collagen
  • Protein-Tyrosine Kinases
  • Calcium