Expansion of mucin-reactive T-helper lymphocytes from patients with colorectal cancer

Cancer Biother. 1995 Summer;10(2):115-23. doi: 10.1089/cbr.1995.10.115.

Abstract

The ability to identify and expand effector cells with reactivity against tumor-associated antigens (TAA) is critical for effective adoptive cellular therapy. The purpose of this study was to assess lymph node lymphocytes sensitized in vivo to the shed TAA TAG-72 as a potential source of cells for adoptive cellular therapy. Lymph nodes containing microscopic tumor and/or shed TAG-72+ mucin were localized using radiolabeled CC49 monoclonal antibody and a gamma detector at the time of exploratory colorectal surgery. Lymph nodes containing microscopic tumor and shed mucin exhibited approximately 40-fold expansion in short-term (< 21 days) cultures with either IL-2 or IL-1 plus IL-2; the combination of IL-2/anti-CD3 monoclonal antibody (mAb) resulted in significantly higher expansion. Cultures generated with IL-2 alone favored the expansion of CD8+ and CD56+ cells, whereas addition of IL-1 or anti-CD3 mAb to IL-2 promoted outgrowth of CD4+ T-cells. The IL-2/anti-CD3 expanded cells exhibited low levels of cytolytic activity in vitro against autologous and allogeneic colon tumor targets. However, CD4+ cells expanded in IL-2/anti-CD3 retained the ability to proliferate in response to TAG-72 mucin-expressing autologous tumor as well as bovine submaxillary mucin (BSM) a soluble TAG-72+ mucin. In addition, CD4+ cells expressed mRNA for IL-2, IL-4, tumor necrosis factor-beta and IFNg, and retained the ability to secrete IL-2 after expansion. Thus, noncytolytic, cytokine-secreting, mucin-reactive T- cells can be expanded from lymph nodes of patients with colorectal cancer.(ABSTRACT TRUNCATED AT 250 WORDS)

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antigens, Neoplasm / analysis*
  • CD4-CD8 Ratio
  • Colorectal Neoplasms / immunology*
  • Colorectal Neoplasms / therapy
  • Cytotoxicity, Immunologic
  • Glycoproteins / analysis*
  • Humans
  • Immunotherapy, Adoptive
  • Interleukin-2 / biosynthesis
  • Lymph Nodes / immunology*
  • Mucins / analysis*
  • Phenotype
  • RNA, Messenger / analysis
  • T-Lymphocytes, Helper-Inducer / immunology*
  • Tumor Cells, Cultured

Substances

  • Antigens, Neoplasm
  • Glycoproteins
  • Interleukin-2
  • Mucins
  • RNA, Messenger
  • tumor-associated antigen 72