Control of beta 3-adrenergic receptor gene expression in brown adipocytes in culture

Mol Cell Endocrinol. 1995 Apr 1;109(2):189-95. doi: 10.1016/0303-7207(95)03502-x.

Abstract

Brown adipose tissue is a mammalian thermogenic tissue. Its ability to dissipate energy as heat is due to a unique mitochondrial protein, uncoupling protein (UCP). Activation and expression of UCP is under control of the sympathetic nervous system acting through beta -adrenergic receptors (AR). In this study we used Siberian hamster brown adipocytes differentiated in vitro to investigate the expression of the fat specific beta 3-AR. Binding studies using the new labelled beta 3 adrenergic ligand [3H]SB 206606 showed a density of beta 3-AR in brown adipocyte plasma membranes comparable to that measured in vivo. beta 3-AR mRNA expression was very high in mature brown adipocytes and was started to be expressed during differentiation before UCP mRNA. Its half-life was approximately 50 min. Treatment of cells with non-specific beta adrenergic agonists, specific beta 3-adrenergic agonists, and dibutyryl cyclic AMP resulted in a marked down regulation of beta 3-AR mRNA level within several hours.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adipose Tissue, Brown / cytology
  • Adipose Tissue, Brown / drug effects
  • Adipose Tissue, Brown / metabolism*
  • Adrenergic beta-Agonists / pharmacology*
  • Animals
  • Base Sequence
  • Cell Differentiation
  • Cells, Cultured
  • Cricetinae
  • Ethanolamines / pharmacology*
  • Gene Expression Regulation / drug effects*
  • Half-Life
  • Isoproterenol / pharmacology
  • Molecular Sequence Data
  • Phodopus
  • Receptors, Adrenergic, beta / biosynthesis
  • Receptors, Adrenergic, beta / genetics*
  • Receptors, Adrenergic, beta-3

Substances

  • Adrenergic beta-Agonists
  • Ethanolamines
  • Receptors, Adrenergic, beta
  • Receptors, Adrenergic, beta-3
  • BRL 37344
  • Isoproterenol