Mechanism of histamine-induced calcium efflux from cultured bovine adrenal chromaffin cells: possible involvement of an Na+/Ca2+ exchange mechanism

Neurosci Lett. 1994 Oct 24;180(2):281-4. doi: 10.1016/0304-3940(94)90539-8.

Abstract

The effect of stimulation of the histamine receptor on Ca2+ mobilization in cultured bovine adrenal chromaffin cells was examined. Histamine (10(-5) M) increased the intracellular free Ca2+ ([Ca2+]i) to a peak in the presence or absence of extracellular Ca2+, followed by decrease with time. Histamine (10(-8)-10(-5) M) also stimulated 45Ca2+ efflux from cultured bovine adrenal chromaffin cells in a concentration dependent manner. Its stimulatory effect on 45Ca2+ efflux was inhibited by the specific histamine H1 receptor antagonist mepyramine. The increase in histamine-stimulated 45Ca2+ efflux was inhibited by deprivation of extracellular Na+ and by the Na+/Ca2+ exchange inhibitor amiloride. In addition, histamine stimulated 22Na+ influx into the cells, and this action was inhibited by amiloride. These results suggest that stimulation of the histamine H1 receptor regulates Na+/Ca2+ exchange in cultured bovine adrenal chromaffin cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adrenal Medulla / drug effects*
  • Adrenal Medulla / metabolism
  • Amiloride / pharmacology
  • Animals
  • Calcium / metabolism*
  • Carrier Proteins / antagonists & inhibitors
  • Carrier Proteins / metabolism*
  • Cattle
  • Dose-Response Relationship, Drug
  • Extracellular Space / metabolism
  • Histamine / pharmacology*
  • Intracellular Fluid / metabolism
  • Pyrilamine / pharmacology
  • Receptors, Histamine H1 / physiology
  • Signal Transduction
  • Sodium / metabolism*
  • Sodium-Calcium Exchanger

Substances

  • Carrier Proteins
  • Receptors, Histamine H1
  • Sodium-Calcium Exchanger
  • Amiloride
  • Histamine
  • Sodium
  • Pyrilamine
  • Calcium