Infrequent somatic mutation of the MTS1 gene in primary bladder carcinomas

Jpn J Cancer Res. 1995 Mar;86(3):249-51. doi: 10.1111/j.1349-7006.1995.tb03047.x.

Abstract

We examined a candidate tumor suppressor gene on chromosome 9p21, MTS1/CDK4I (multiple tumor suppressor 1/cyclin-dependent kinase 4 inhibitor), which has been found to be mutated frequently in cell lines derived from bladder carcinomas, for somatic mutations in 39 primary bladder cancers by means of SSCP (single-stranded conformational polymorphism) and DNA sequencing. Mutations were detected in two of these carcinomas; one was a 61-base deletion and the other a 1-base deletion. In both cases the homologous allele was missing, indicating that "two-hit" mutation of the MTS1 gene had taken place in these tumors. The results indicated that inactivation of the MTS1 gene is likely to be a contributing factor in some, but not the majority of, bladder cancers.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • Carrier Proteins / genetics*
  • Chromosomes, Human, Pair 9*
  • Cyclin-Dependent Kinase Inhibitor p16
  • Gene Deletion
  • Humans
  • Molecular Sequence Data
  • Mutation*
  • Polymorphism, Single-Stranded Conformational
  • Sequence Analysis, DNA
  • Urinary Bladder Neoplasms / genetics*

Substances

  • Carrier Proteins
  • Cyclin-Dependent Kinase Inhibitor p16