Inhibition of interleukin-1-induced effects in synoviocytes transduced with the human IL-1 receptor antagonist cDNA using an adenoviral vector

Hum Gene Ther. 1995 Mar;6(3):307-16. doi: 10.1089/hum.1995.6.3-307.

Abstract

In this report, we present data showing that a recombinant adenoviral vector (Ad.RSVIL-1ra) containing the cDNA for human interleukin-1 receptor antagonist protein (IL-1ra) can genetically modify synoviocytes both in vitro and in vivo. Human synoviocytes infected with Ad.RSVIL-1ra in vitro expressed and secreted high levels of human IL-1ra that were detected by ELISA of tissue culture supernatants. New Zealand White rabbits that received intra-articular injections of Ad.RSVIL-1ra expressed transgenic IL-1ra in synoviocytes, and secretion was detected for at least 4 weeks post-infection. Further, biological activity of the transgenic IL-1ra was demonstrated by its ability to inhibit IL-1-induced prostaglandin E2 (PGE2) synthesis in vitro and IL-1-induced glycosaminoglycan (GAG) degradation in vivo. These data demonstrate that recombinant adenoviral vectors can mediate the intra-articular expression of anti-inflammatory proteins and may be a reasonable method to deliver therapeutically relevant proteins for the regional treatment of synovial inflammation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adenoviridae / genetics*
  • Animals
  • Anti-Inflammatory Agents / pharmacology
  • Antisense Elements (Genetics)
  • Blotting, Southern
  • Cell Line, Transformed / virology
  • Cells, Cultured / virology
  • DNA / analysis
  • DNA / chemistry
  • DNA, Complementary / chemistry
  • DNA, Complementary / genetics
  • Dinoprostone / antagonists & inhibitors
  • Dinoprostone / biosynthesis
  • Enzyme-Linked Immunosorbent Assay
  • Genetic Vectors / chemistry*
  • Genetic Vectors / genetics*
  • Humans
  • Interleukin 1 Receptor Antagonist Protein
  • Interleukin-1 / pharmacology
  • Knee Joint
  • Rabbits
  • Receptors, Interleukin-1 / antagonists & inhibitors*
  • Recombinant Proteins / pharmacology
  • Sialoglycoproteins / genetics*
  • Sialoglycoproteins / metabolism
  • Sialoglycoproteins / pharmacology
  • Synovial Membrane / cytology
  • Synovial Membrane / drug effects*
  • Synovial Membrane / virology*
  • Therapeutic Irrigation
  • Time Factors
  • Transduction, Genetic

Substances

  • Anti-Inflammatory Agents
  • Antisense Elements (Genetics)
  • DNA, Complementary
  • IL1RN protein, human
  • Interleukin 1 Receptor Antagonist Protein
  • Interleukin-1
  • Receptors, Interleukin-1
  • Recombinant Proteins
  • Sialoglycoproteins
  • DNA
  • Dinoprostone