Esophageal varices

Gastrointest Endosc Clin N Am. 1994 Oct;4(4):747-71.

Abstract

Numerous conditions lead to portal hypertension with the development of esophageal varices. Treatment for acute variceal hemorrhage should progress in a logical, stepwise fashion. Therapy after fluid resuscitation includes vasopressin, somatostatin, or a Sengstaken-Blakemore tube. This is followed by treatment with sclerotherapy, variceal ligation, or a combination of both. Continued bleeding is managed by more invasive measures that include radiologic embolization or shunting, esophageal transection, distal splenorenal shunt, or liver transplantation. Beta-blockade may be useful to prevent recurrent bleeding in compliant patients without medical conditions that would preclude use of beta-blockade. Once control of the bleeding has been achieved, sclerotherapy or ligation should be used to obliterate the varices, but prophylactic use of sclerosant is not particularly beneficial.

Publication types

  • Review

MeSH terms

  • Adrenergic beta-Antagonists / therapeutic use
  • Combined Modality Therapy
  • Embolization, Therapeutic / methods
  • Esophageal and Gastric Varices / complications
  • Esophageal and Gastric Varices / therapy*
  • Esophagoscopy
  • Gastrointestinal Hemorrhage / etiology
  • Gastrointestinal Hemorrhage / therapy*
  • Humans
  • Intubation, Gastrointestinal / methods
  • Ligation / methods
  • Liver Transplantation
  • Portasystemic Shunt, Surgical / methods
  • Randomized Controlled Trials as Topic
  • Sclerotherapy / methods
  • Somatostatin / therapeutic use
  • Vasopressins / therapeutic use

Substances

  • Adrenergic beta-Antagonists
  • Vasopressins
  • Somatostatin