Competitive inhibition of HIV-1 protease by biphenyl carboxylic acids

Arch Biochem Biophys. 1995 Jan 10;316(1):523-8. doi: 10.1006/abbi.1995.1069.

Abstract

A novel series of nonpeptidic compounds that contain a biphenyl carboxylic acid group have been shown to inhibit HIV-1 protease. The active compounds, most of which are highly soluble, have IC50 values in the range of 3.4-74 microM. The structure-inhibitory activity relationship demonstrates the necessity of the biphenyl carboxylic acid group for inhibition, which is enhanced by the presence of a sulfone group and by halogenation of an adjacent phenyl group. A double reciprocal plot of inhibition data on two of the compounds clearly shows that the inhibition occurs in a competitive manner, with Ki values of 1.1 and 3.4 microM. Inhibition by several of the compounds was found to be reversible and fast-binding, while one of the biphenyl carboxylic acids inhibits in a reversible slow-binding manner. Time-dependent inhibition studies were conducted on this compound, and it was determined to have the kinetic values of kon = 0.18 microM-1min-1, koff = 9.7 x 10(-2)min-1, and Ki = 0.14 microM. Thus, the slow-binding inhibitor is the most potent in the series. Molecular modeling has provided information on a possible binding mode for two different biphenyl carboxylic acid inhibitors of HIV-1 protease.

MeSH terms

  • Amino Acid Sequence
  • Binding Sites
  • Biphenyl Compounds / chemistry
  • Biphenyl Compounds / pharmacology*
  • Carboxylic Acids / chemistry
  • Carboxylic Acids / pharmacology*
  • Computer Simulation
  • Drug Design
  • HIV Protease Inhibitors / chemistry
  • HIV Protease Inhibitors / classification
  • HIV Protease Inhibitors / pharmacology*
  • HIV-1 / enzymology*
  • Kinetics
  • Models, Molecular
  • Molecular Sequence Data
  • Recombinant Proteins / drug effects
  • Solubility

Substances

  • Biphenyl Compounds
  • Carboxylic Acids
  • HIV Protease Inhibitors
  • Recombinant Proteins