Overexpression of human immunodeficiency virus type 1 protease increases intracellular cleavage of Gag and reduces virus infectivity

Virology. 1995 Feb 1;206(2):854-65. doi: 10.1006/viro.1995.1008.

Abstract

Here we have investigated if human immunodeficiency virus type 1 (HIV-1) protease expressed in trans can interfere with production of infectious HIV-1 particles. Protease produced from a Tat and Rev inducible expression plasmid specifically cleaved HIV-1 p55Gag in a dose-dependent manner. Coexpression of protease and an infectious HIV-1 proviral clone resulted in increased intracellular cleavage of p55Gag. As a consequence, virus production and virus infectivity was significantly reduced. These results suggest that overexpression of HIV-1 protease in HIV-1-infected cells is a powerful way to inhibit production of infectious virions.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • Cell Line
  • Cells, Cultured
  • DNA Primers
  • Gene Expression
  • Gene Products, gag / metabolism*
  • HIV Core Protein p24 / biosynthesis
  • HIV Protease / biosynthesis*
  • HIV Protease / metabolism
  • HIV-1 / pathogenicity
  • HIV-1 / physiology*
  • Humans
  • Kinetics
  • Leukocytes, Mononuclear / virology
  • Molecular Sequence Data
  • Mutagenesis, Site-Directed
  • Plasmids
  • Polymerase Chain Reaction
  • Recombinant Proteins / biosynthesis
  • Recombinant Proteins / metabolism
  • Transfection
  • Virus Replication*

Substances

  • DNA Primers
  • Gene Products, gag
  • HIV Core Protein p24
  • Recombinant Proteins
  • HIV Protease