Modulation of lipid metabolism at rat hepatic subcellular sites by female sex hormones

Biochim Biophys Acta. 1995 Feb 9;1254(3):267-73. doi: 10.1016/0005-2760(94)00187-4.

Abstract

The present studies examine the modulation of lipoprotein metabolism at subcellular sites in the liver by female sex hormones. Subcutaneous injection of ethinyl estradiol (5 mg/kg) decreased both triacylglycerol (TG) lipase activity and neutral cholesteryl ester (CE) hydrolase activity in hepatic endosomes while increasing lysosomal lipid hydrolysis. These data suggest that estrogen may induce a shift in the site of intracellular lipid catabolism similar to that found in fasting animals [1]. This work also shows that TG-lipase activity is increased in the CURL and MVB endosomal fractions of ovariectomized rats compared to that found in the equivalent endosomal compartments of age-matched intact female controls. These observations are consistent with an inhibition of endosomal lipase by female sex hormones under physiologic conditions.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acetylglucosaminidase / metabolism
  • Animals
  • Coronary Disease / prevention & control
  • Endosomes / metabolism
  • Ethinyl Estradiol / pharmacology*
  • Female
  • Lipase / metabolism
  • Lipid Metabolism*
  • Lipoproteins / metabolism*
  • Liver / drug effects
  • Liver / metabolism*
  • Liver / ultrastructure
  • Lysosomes / metabolism
  • Ovariectomy
  • Rats
  • Rats, Sprague-Dawley
  • Sterol Esterase / metabolism

Substances

  • Lipoproteins
  • Ethinyl Estradiol
  • Sterol Esterase
  • Lipase
  • Acetylglucosaminidase