Potassium-induced depolarization stimulates somatostatin gene expression in cultured fetal rat cerebrocortical cells

J Pediatr Endocrinol. 1993 Jul-Dec;6(3-4):219-23. doi: 10.1515/jpem.1993.6.3-4.219.

Abstract

The stimulatory effect of potassium depolarization upon somatostatin mRNA (SS mRNA) levels in primary cultures of fetal cerebrocortical cells was analyzed. Depolarizing stimuli, such as 56 mM K+ concentration for 24 hours, elicited an increase in immunoreactive somatostatin (IR-SS) release to the media and SS mRNA levels, suggesting that somatostatin secretion can be coupled to SS mRNA accumulation. These changes were inhibited by the Ca2+ channel antagonist verapamil (VPM). In contrast, Na+ channel blockade by tetrodotoxin (TTX) did not modify the 24 h potassium-induced increase in SS mRNA. These results suggest that the induction of SS mRNA expression by K+ involves the modulation of calcium ion channels.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cells, Cultured
  • Cerebral Cortex / cytology
  • Cerebral Cortex / embryology*
  • Electrophysiology
  • Fetus / cytology
  • Fetus / physiology*
  • Gene Expression / drug effects*
  • Potassium / pharmacology*
  • RNA, Messenger / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Somatostatin / genetics*
  • Somatostatin / metabolism

Substances

  • RNA, Messenger
  • Somatostatin
  • Potassium