Acetaldehyde regulates the gene expression of matrix-metalloproteinase-1 and -2 in human fat-storing cells

Life Sci. 1994;55(17):1311-6. doi: 10.1016/0024-3205(94)00763-2.

Abstract

Altered degradation of extracellular matrix has been implicated in the pathogenesis of hepatic fibrosis. We studied the effect of acetaldehyde (AcCHO) on gene expression of matrix-metalloproteinase (MMP)-1 (fibroblast type- interstitial collagenase) and MMP-2 (72 kDa gelatinase-type IV collagenase) in comparison with the AcCHO effect on collagen type I and IV synthesis in cultures of fat-storing cells (FSC) isolated from normal human livers. Cultured human FSC expressed single mRNA transcripts (2.7 and 3.2 kb) specific for MMP-1 and MMP-2, respectively. AcCHO inhibited MMP-1 mRNA levels, whereas it stimulated collagen type I mRNA and protein expression. Opposite AcCHO effects were evident on MMP-2 mRNA and collagen IV synthesis, being MMP-2 up-regulated and collagen IV down-regulated. These data suggest that regulation of MMP-1 and MMP-2 genes by AcCHO may contribute to disruption of the normal basement membrane and its replacement with fibrillar collagens in the early stages of alcoholic liver fibrosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetaldehyde / pharmacology*
  • Cells, Cultured
  • Collagen / biosynthesis
  • Collagenases / genetics*
  • Extracellular Matrix / enzymology*
  • Gene Expression Regulation, Enzymologic / drug effects*
  • Humans
  • Kinetics
  • Lipid Metabolism
  • Liver / cytology
  • Liver / enzymology*
  • Liver / metabolism
  • Matrix Metalloproteinase 1
  • Matrix Metalloproteinase 9
  • Metalloendopeptidases / genetics*
  • RNA, Messenger / metabolism

Substances

  • RNA, Messenger
  • Collagen
  • Collagenases
  • Metalloendopeptidases
  • Matrix Metalloproteinase 9
  • Matrix Metalloproteinase 1
  • Acetaldehyde