Vascular expression of the grp1.8 promoter is controlled by three specific regulatory elements and one unspecific activating sequence

Plant Mol Biol. 1994 Oct;26(2):747-56. doi: 10.1007/BF00013759.

Abstract

The bean grp1.8 full-length promoter is specifically active in vascular tissue during normal development of tobacco. Deletion of a negative regulatory element resulted in ectopic activity of the promoter in cortical cells of hypocotyls, roots and stems. A 169 bp fragment (-205 to -36) of the grp1.8 promoter conferred vascular-specific expression to CaMV 35S minimal promoters whereas a 141 bp fragment (-205 to -64) strongly activated these minimal promoters both in vascular and cortical cells. These experiments defined a new regulatory element (VSE) that is essential for vascular-specific expression and is located between -64 and -36. The 141 bp grp1.8 promoter sequence had enhancer-like properties as it was active in both orientations. A 24 bp sequence (bp -119 to -96, corresponding to the SE1 regulatory element) enhanced expression from several minimal promoters strongly but unspecifically, whereas a 26 bp sequence (-98 to -73, corresponding to the RSE regulatory element) induced vascular-specific expression. Thus, the grp1.8 promoter is regulated by a combinatorial mechanism that can integrate the action of different, non-additively acting regulatory elements into vascular-specific expression.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • Escherichia coli / genetics
  • Gene Expression
  • Gene Expression Regulation, Plant*
  • Glucuronidase / biosynthesis
  • Molecular Sequence Data
  • Nicotiana / genetics*
  • Nicotiana / growth & development
  • Oligodeoxyribonucleotides
  • Plants, Genetically Modified
  • Plants, Toxic*
  • Plasmids
  • Promoter Regions, Genetic*
  • Regulatory Sequences, Nucleic Acid*
  • Restriction Mapping
  • Rhizobium / genetics
  • Transformation, Genetic
  • Vacuoles / metabolism*

Substances

  • Oligodeoxyribonucleotides
  • Glucuronidase