Oligonucleotides protect cells from the cytotoxicity of several anti-cancer chemotherapeutic drugs

Anticancer Drugs. 1994 Aug;5(4):437-42.

Abstract

The possibility of inhibiting gene expression with antisense oligonucleotides (AS ODNs) in combination with more conventional chemotherapy is a very attractive modality in oncology. However, possible interaction between the ODN and drug must be considered. Here we show that ODNs protect cells from the cytostatic/cytotoxic action of actinomycin D (AMD), adriamycin, daunomycin or quinacrine, but not mitomycin, camptothecin, vincristine, cisplatin, etoposide (VP-16) or cycloheximide. The cytoprotective effect depends on ODN length as well as ability to interact directly with the cytotoxic drug and is only slightly sequence selective.

Publication types

  • Comparative Study

MeSH terms

  • Animals
  • Antineoplastic Agents / metabolism
  • Antineoplastic Agents / toxicity*
  • Base Sequence
  • Cell Survival / drug effects
  • Dactinomycin / metabolism
  • Dactinomycin / toxicity
  • Daunorubicin / metabolism
  • Daunorubicin / toxicity
  • Doxorubicin / metabolism
  • Doxorubicin / toxicity
  • Drug Interactions
  • Humans
  • Molecular Sequence Data
  • Oligonucleotides, Antisense / metabolism
  • Oligonucleotides, Antisense / therapeutic use*
  • Receptors, Tumor Necrosis Factor / drug effects

Substances

  • Antineoplastic Agents
  • Oligonucleotides, Antisense
  • Receptors, Tumor Necrosis Factor
  • Dactinomycin
  • Doxorubicin
  • Daunorubicin