Pharmacological inhibition of programmed lymphocyte death

Immunol Today. 1994 May;15(5):235-42. doi: 10.1016/0167-5699(94)90249-6.

Abstract

Programmed cell death (PCD) is a necessary process that helps to regulate the lifespan of lymphocytes and maintain the compartmental balance of lymphoid organs. In addition, PCD is required for the generation and maintenance of self-tolerance. Strategies that inhibit PCD cause profound alterations in the (patho)physiology of the immune system. Here, Guido Kroemer and Carlos Martínez-A. discuss the multiplicity of PCD-inducing pathways, which have been revealed through the use of PCD-inhibitory agents, and analyse the levels at which these agents act.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Apoptosis / drug effects*
  • Cycloheximide / pharmacology
  • Cyclosporine / pharmacology
  • Humans
  • Hydroxyquinolines / pharmacology
  • Lymphocytes / drug effects*
  • Lymphocytes / physiology
  • Mifepristone / pharmacology
  • Proto-Oncogenes / physiology
  • Tretinoin / pharmacology

Substances

  • Hydroxyquinolines
  • Mifepristone
  • roquinimex
  • Tretinoin
  • Cyclosporine
  • Cycloheximide