Free protein S deficiency in patients with Crohn's disease

Scand J Gastroenterol. 1994 Apr;29(4):333-5. doi: 10.3109/00365529409094844.

Abstract

Multifocal intestinal infarctions, due to thrombosis in small vessels, might be a pathogenetic mechanism for Crohn's disease (CD). Deficiency of free protein S may contribute to the development of such thrombotic occlusions. In the present study free protein S was measured in 54 patients with CD. In 31 patients (57.4%) the plasma concentrations of free protein S were below the lower normal range. The mean value of free protein S in CD patients was 72.2%, as compared with 97.5% in healthy subjects (p < 0.01). The concentrations of C4b-binding protein and protein C were similar in the two groups. Free protein S levels were not correlated to disease activity, previous surgery or complications, extraintestinal manifestations, or current medical therapy. The impairment of the protein S/protein C/thrombomodulin system found in patients with CD favours coagulation and might be of importance for both the development of CD and its thromboembolic complications.

MeSH terms

  • Adult
  • Aged
  • Carrier Proteins / metabolism
  • Complement Inactivator Proteins*
  • Crohn Disease / etiology*
  • Crohn Disease / metabolism
  • Female
  • Glycoproteins*
  • Humans
  • Infarction / complications
  • Infarction / metabolism
  • Intestines / blood supply
  • Male
  • Middle Aged
  • Protein S Deficiency*
  • Thromboembolism / complications
  • Thromboembolism / metabolism

Substances

  • Carrier Proteins
  • Complement Inactivator Proteins
  • Glycoproteins