Transfection of the bcr/abl oncogene into factor-dependent cells by electroporation: acquisition of autonomous proliferation

Hematol Oncol. 1994 Mar-Apr;12(2):53-60. doi: 10.1002/hon.2900120202.

Abstract

In order to clarify the function of P210 bcr/abl oncogene in leukemogenesis, IL-3 dependent murine hematopietic cell line, FDC-P2, was transfected with the plasmid containing cDNA of P210 bcr/abl oncogene (pGD'210) or murine IL-3 (pcDmIL3) by electroporation. Four out of five pGDH210 transfected clones as well as FDC-P2 transfected with pcDmIL3, acquired autonomous proliferation (i.e. lost the requirement for IL-3 supplementation). The expression of bcr/abl oncogene was weak in one clone, which remained dependent on IL-3. Unlike pcDmIL3 transfectants, which secrete IL-3 into the supernatant, IL-3 was not demonstrated in the culture supernatant of pGD'210 transfected FDC-P2. These finding suggest that P210 bcr/abl oncogene is directly associated with autonomous proliferation, which is the first process of leukemogenesis.

MeSH terms

  • Animals
  • Base Sequence
  • Cell Division*
  • Cell Line
  • Culture Media, Conditioned
  • DNA Primers
  • Electroporation / methods
  • Fusion Proteins, bcr-abl / biosynthesis*
  • Genes, abl*
  • Hematopoietic Stem Cells
  • Interleukin-3 / biosynthesis
  • Mice
  • Molecular Sequence Data
  • Oncogenes*
  • Plasmids
  • Polymerase Chain Reaction
  • Transfection*

Substances

  • Culture Media, Conditioned
  • DNA Primers
  • Interleukin-3
  • Fusion Proteins, bcr-abl