Translocation (3;21)(q26;q22) in therapy-related myelodysplasia following drugs targeting DNA-topoisomerase II combined with alkylating agents, and in myeloproliferative disorders undergoing spontaneous leukemic transformation

Cancer Genet Cytogenet. 1994 Aug;76(1):50-5. doi: 10.1016/0165-4608(94)90071-x.

Abstract

Translocation (3;21)(q26;q22) has been observed only rarely in de novo myelodysplasia (MDS) and de novo acute myeloid leukemia (AML), but, including the two new cases in the present study, the aberration has now been identified in at least 10 cases of t-MDS or t-AML. All these 10 patients had previously received alkylating agents, in nine patients combined with a drug targeting at DNA-topoisomerase II (doxorubicin in eight cases). Eight of the ten patients presented with t-MDS. A further 20 patients with various myeloproliferative disorders and an identical t(3;21) have been reported. In these cases, t(3;21) was not related to any specific type of previous therapy but was associated with transformation from chronic stage disease to overt AML.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Alkylating Agents / adverse effects*
  • Antineoplastic Combined Chemotherapy Protocols / adverse effects
  • Chromosomes, Human, Pair 21*
  • Chromosomes, Human, Pair 3*
  • Humans
  • Karyotyping
  • Leukemia, Myeloid / genetics*
  • Myelodysplastic Syndromes / chemically induced
  • Myelodysplastic Syndromes / genetics*
  • Myeloproliferative Disorders / genetics*
  • Myeloproliferative Disorders / pathology
  • Topoisomerase II Inhibitors*
  • Translocation, Genetic*

Substances

  • Alkylating Agents
  • Topoisomerase II Inhibitors