Transforming growth factor-alpha and endocrine sensitivity in breast cancer

Cancer Res. 1994 Apr 1;54(7):1684-9.

Abstract

The expression of transforming growth factor-alpha (TGF-alpha) has been evaluated in 51 breast cancers of known responsiveness to endocrine therapy using immunohistochemistry. High levels of TGF-alpha were observed in 65% of tumors and showed no relationship with tumor estrogen receptor or epidermal growth factor receptor status or Ki67 immunostaining. TGF-alpha levels did, however, relate to the endocrine sensitivity of the disease, with unresponsive tumors frequently showing high levels of TGF-alpha immunoreactivity. This relationship was observed in estrogen receptor-positive disease and was independent of the epidermal growth factor receptor status of the tumor. No quantitative association between TGF-alpha and Ki67 immunostaining was observed in any of the subcategories of tumors. These data infer a role for TGF-alpha in the development of endocrine insensitivity in estrogen receptor-positive breast cancer by mechanisms which appear independent of tumor growth fraction, as determined by Ki67 immunostaining.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Biomarkers, Tumor / analysis*
  • Breast Neoplasms / drug therapy
  • Breast Neoplasms / pathology*
  • ErbB Receptors / analysis
  • Female
  • Goserelin / therapeutic use*
  • Humans
  • Immunohistochemistry
  • Ki-67 Antigen
  • Menopause
  • Neoplasm Proteins / analysis*
  • Nuclear Proteins / analysis*
  • Receptors, Estrogen / analysis
  • Tamoxifen / therapeutic use*
  • Transforming Growth Factor alpha / analysis*
  • Transforming Growth Factor alpha / biosynthesis

Substances

  • Biomarkers, Tumor
  • Ki-67 Antigen
  • Neoplasm Proteins
  • Nuclear Proteins
  • Receptors, Estrogen
  • Transforming Growth Factor alpha
  • Tamoxifen
  • Goserelin
  • ErbB Receptors