Regulation of rat-kidney cortex fructose-1,6-bisphosphatase activity. II. Effects of adenine nucleotides

Int J Biochem. 1993 Dec;25(12):1969-74. doi: 10.1016/0020-711x(88)90333-3.

Abstract

1. The native rat-kidney cortex Fructose-1,6-bisphosphatase is differentially regulated by adenine nucleotides in the presence of divalent cations. 2. Binding of AMP and ADP to the enzyme is co-operative. The inhibition by both nucleotides show an uncompetitive mechanism AMP being the most efficient inhibitor. 3. Mg2+ decreases the inhibition produced by AMP and ADP by enhancing their I0.5 and completely annulates the inhibitory effect of ATP. 4. In the presence of Mn2+ ADP behaves as an inhibitor but no inhibition is evident with AMP, suggesting the existence of different allosteric sites for each nucleotide.

MeSH terms

  • Adenine Nucleotides / pharmacology*
  • Adenosine Diphosphate / pharmacology
  • Adenosine Monophosphate / pharmacology
  • Adenosine Triphosphate / pharmacology
  • Animals
  • Fructose-Bisphosphatase / antagonists & inhibitors
  • Fructose-Bisphosphatase / drug effects*
  • Kidney Cortex / drug effects*
  • Kidney Cortex / enzymology
  • Magnesium / pharmacology*
  • Manganese / pharmacology*
  • Rats

Substances

  • Adenine Nucleotides
  • Adenosine Monophosphate
  • Manganese
  • Adenosine Diphosphate
  • Adenosine Triphosphate
  • Fructose-Bisphosphatase
  • Magnesium