Human immunodeficiency virus type 1 Nef protein down-regulates transcription factors NF-kappa B and AP-1 in human T cells in vitro after T-cell receptor stimulation

J Virol. 1994 May;68(5):3243-9. doi: 10.1128/JVI.68.5.3243-3249.1994.

Abstract

Human immunodeficiency virus type 1 (HIV-1) negative factor (Nef) has been shown to down-regulate the transcription factors NF-kappa B and AP-1 in vitro. To define the mechanism of action of the Nef protein, the signal transduction pathways which may be affected in T cells by constitutive expression of the nef gene were examined. Stimulation of T cells with tumor necrosis factor, interleukin-1, or lipopolysaccharide resulted in the recruitment of transcriptional factors to a similar level whether or not the cells expressed the nef gene. On the other hand, stimulation of T cells by mitogens or antibodies to the T-cell receptor (TCR)-CD3 complex resulted in the down-regulation of transcriptional factors NF-kappa B and AP-1 in cells expressing the nef gene compared with cells not expressing the nef gene. Because the Nef protein does not affect the surface expression of the CD3-TCR complex, we conclude that the Nef protein down-regulates the transcriptional factors NF-kappa B and AP-1 in T cells in vitro through an effect on the TCR-dependent signal transduction pathway.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Antigens, CD / metabolism
  • Base Sequence
  • Down-Regulation
  • Gene Products, nef / pharmacology*
  • HIV-1 / genetics*
  • Humans
  • Lymphocyte Activation
  • Molecular Sequence Data
  • NF-kappa B / drug effects
  • Phorbols / pharmacology
  • Phytohemagglutinins / pharmacology
  • Proto-Oncogene Proteins c-jun / drug effects
  • Receptors, Cell Surface / metabolism
  • Receptors, Cytokine / metabolism
  • Signal Transduction*
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / metabolism*
  • Transcription Factors / drug effects*
  • nef Gene Products, Human Immunodeficiency Virus

Substances

  • Antigens, CD
  • Gene Products, nef
  • NF-kappa B
  • Phorbols
  • Phytohemagglutinins
  • Proto-Oncogene Proteins c-jun
  • Receptors, Cell Surface
  • Receptors, Cytokine
  • Transcription Factors
  • nef Gene Products, Human Immunodeficiency Virus
  • phorbol