Suppression of Philadelphia1 leukemia cell growth in mice by BCR-ABL antisense oligodeoxynucleotide

Proc Natl Acad Sci U S A. 1994 May 10;91(10):4504-8. doi: 10.1073/pnas.91.10.4504.

Abstract

When injected into SCID mice, the Philadelphia chromosome-positive chronic myeloid leukemia-blast crisis cell line BV173 induces a disease process closely resembling that seen in leukemia patients. At 1 and 3 weeks after injection of 10(6) BV173 cells, CD10+ cells were detected in the bone marrow of the mice, leukemic colonies grew from bone marrow and spleen cell suspensions, and BCR-ABL transcripts were detectable in bone marrow, spleen, peripheral blood, liver, and lungs. Systemic treatment of the leukemic mice with a 26-mer BCR-ABL antisense oligodeoxynucleotide (1 mg/day for 9 days) induced disappearance of CD10+ and clonogenic leukemic cells and a marked decrease in BCR-ABL mRNA in mouse tissues. Untreated mice or mice treated with a BCR-ABL sense oligodeoxynucleotide or a 6-base-mismatched antisense oligodeoxynucleotide oligodeoxynucleotide were dead 8-13 weeks after leukemia cell injection; in marked contrast, mice treated with BCR-ABL antisense oligodeoxynucleotide died of leukemia 18-23 weeks after injection of leukemic cells. These findings provide evidence for the in vivo effectiveness of an anticancer therapy based on antisense oligodeoxynucleotides targeting a tumor-specific gene.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Base Sequence
  • Cell Division / drug effects*
  • Cell Line
  • Flow Cytometry
  • Fusion Proteins, bcr-abl / biosynthesis
  • Fusion Proteins, bcr-abl / genetics*
  • Genes, abl
  • Humans
  • Kidney / pathology
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / drug therapy*
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / pathology*
  • Leukemia, Promyelocytic, Acute / pathology
  • Liver / pathology
  • Male
  • Mice
  • Mice, SCID
  • Molecular Sequence Data
  • Oligodeoxyribonucleotides / toxicity
  • Oligonucleotides, Antisense / analysis
  • Oligonucleotides, Antisense / therapeutic use*
  • Oligonucleotides, Antisense / toxicity*
  • Spleen / pathology
  • Time Factors
  • Transcription, Genetic
  • Translocation, Genetic
  • Transplantation, Heterologous
  • Tumor Cells, Cultured

Substances

  • Oligodeoxyribonucleotides
  • Oligonucleotides, Antisense
  • Fusion Proteins, bcr-abl