TCR antagonism and T cell tolerance can be independently induced in a DR-restricted, hemagglutinin-specific T cell clone

Int Immunol. 1994 Mar;6(3):363-7. doi: 10.1093/intimm/6.3.363.

Abstract

The outcome of TCR engagement with peptide-MHC is of central importance for the immune response of the host. TCR antagonism is one phenomenon known which is characterized by selective inhibition of T cell responses by non-stimulatory antigen analogs. T cell anergy is another state resulting in T cell unresponsiveness, generally characterized by lack of proliferation and lymphokine production. In the present study, the relationship between TCR antagonism and T cell anergy was examined by using protocols known to induce either phenomenon. Re-isolation experiments demonstrated that antagonized T cells were not tolerized, in that they were fully capable of responding to a subsequent antigen challenge. Conversely, while high doses of soluble antigen could efficiently induce T cell tolerance, TCR antagonists, either alone or in conjunction with suboptimal antigen doses, could not. Taken together, these data demonstrate that TCR antagonism and T cell tolerance are phenomena independent of each other.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antigen-Presenting Cells / immunology
  • Antigens / immunology
  • Cell Line, Transformed
  • Clonal Anergy / physiology*
  • Clone Cells
  • HLA-DR Antigens
  • Hemagglutinins / immunology
  • Humans
  • Immune Tolerance / physiology*
  • Peptides / immunology
  • Receptors, Antigen, T-Cell / antagonists & inhibitors*
  • T-Lymphocytes / immunology*

Substances

  • Antigens
  • HLA-DR Antigens
  • Hemagglutinins
  • Peptides
  • Receptors, Antigen, T-Cell