The opioid mechanism of interferon-alpha action

Anticancer Drugs. 1994 Feb;5(1):90-4. doi: 10.1097/00001813-199402000-00014.

Abstract

Rats were trained to discriminate the opioid receptor agonist ethylketocyclazocine (EKC) (0.3 mg/kg body weight, intraperitoneally) from saline. Interferon-alpha (IFN-alpha), when substituted for EKC, elicited a dose-related increase in EKC-like responses. This generalization of EKC responses was blocked by the opioid antagonist naloxone (1 mg/kg). Potentiation of responses to a low dose (0.1 mg/kg) of EKC by IFN-alpha (1 x 10(6) U/kg or 0.22 nmol/kg) was also observed. Data thus indicate the involvement of opioid neurons on the action of IFN-alpha. d-Amphetamine (0.8 mg/kg) was shown to potentiate both EKC (0.1 mg/kg) and IFN-alpha (1 x 10(6) U/kg). The present study confirms our previously proposed opioid-mediated dopaminergic mechanism of IFN-alpha.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Dextroamphetamine / pharmacology
  • Discrimination Learning / drug effects
  • Discrimination, Psychological / drug effects*
  • Dose-Response Relationship, Drug
  • Drug Synergism
  • Ethylketocyclazocine / pharmacology
  • Extinction, Psychological / drug effects
  • Generalization, Psychological
  • Interferon-alpha / antagonists & inhibitors
  • Interferon-alpha / pharmacology*
  • Male
  • Naloxone / pharmacology
  • Rats
  • Rats, Wistar
  • Receptors, Opioid / drug effects*

Substances

  • Interferon-alpha
  • Receptors, Opioid
  • Naloxone
  • Ethylketocyclazocine
  • Dextroamphetamine