Decreased hepatocarcinogenic effect of diethylnitrosamine in experimentally induced liver cirrhosis in rat: delay or inhibition?

Cancer Lett. 1994 Apr 29;79(1):1-7. doi: 10.1016/0304-3835(94)90055-8.

Abstract

The relationship between CCl4 or (CCl4 + phenobarbital)-induced liver cirrhosis and diethylnitrosamine (DEN) hepatocarcinogenesis in male F-344 rats was investigated. DEN given alone produced no liver lesions after 16 weeks, but 4/12 (33%) neoplastic nodules developed when nitrosamine was administered to rats with previously established cirrhosis. On the other hand, (CCl4 + phenobarbital) post-treatment had an even stronger effect, increasing the yield of neoplastic nodules to 100% (28/28). Since the exposure time of DEN was the same in all treated groups (4 months), the results indicate the decreased effectiveness of (CCl4 + phenobarbital) pretreatment on DEN hepatocarcinogenesis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Carbon Tetrachloride / toxicity
  • Carbon Tetrachloride Poisoning / pathology*
  • Diethylnitrosamine / toxicity*
  • Liver / drug effects
  • Liver / pathology*
  • Liver Cirrhosis, Experimental / chemically induced
  • Liver Cirrhosis, Experimental / pathology*
  • Liver Neoplasms, Experimental / chemically induced
  • Liver Neoplasms, Experimental / pathology*
  • Liver Neoplasms, Experimental / prevention & control
  • Male
  • Phenobarbital / toxicity*
  • Precancerous Conditions / chemically induced
  • Precancerous Conditions / pathology*
  • Precancerous Conditions / prevention & control
  • Rats
  • Rats, Inbred F344
  • Time Factors

Substances

  • Diethylnitrosamine
  • Carbon Tetrachloride
  • Phenobarbital