Laminin receptor expression and function in small-cell lung carcinoma

Int J Cancer Suppl. 1994:8:116-20. doi: 10.1002/ijc.2910570725.

Abstract

Interactions between tumor cells and laminin or other components of the extracellular matrix are thought to play a role in tumor invasion and metastasis. To analyze these interactions, we examined the expression of 5 types of laminin receptors on 11 cell lines derived from the highly malignant and metastatic tumor small-cell lung cancer (SCLC). The integrins VLA-1, VLA-3, VLA-6 and the 67 KDa monomeric receptor were expressed at various levels, whereas the VLA-2 receptor was absent on the cell lines. Only one cell line expressed none of these laminin receptors. All cell lines co-expressed alpha 6 beta 1 (VLA-6) and the 67-kDa molecule, the only receptors specific for laminin. Analysis of the ability of SCLC cells to bind radiolabeled laminin and to adhere to laminin substrata revealed a correlation between these 2 parameters and the expression of VLA-6 and the 67-kDa monomeric receptor. Cell adhesion was mediated by alpha 6 beta 1, as indicated by inhibition of adhesion using an anti-VLA-6 monoclonal antibody (MAb). Both VLA-6 and the monomeric receptor were up-regulated in vitro by laminin.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antibodies / pharmacology
  • Antibodies, Monoclonal / pharmacology
  • Carcinoma, Small Cell / metabolism*
  • Cell Adhesion / drug effects
  • Cell Line
  • Fluorescent Antibody Technique
  • Humans
  • Integrins / biosynthesis*
  • Integrins / drug effects
  • Integrins / metabolism
  • Laminin / metabolism*
  • Lung Neoplasms / metabolism*
  • Molecular Weight
  • Receptors, Laminin / biosynthesis*
  • Receptors, Laminin / metabolism
  • Regression Analysis
  • Tumor Cells, Cultured

Substances

  • Antibodies
  • Antibodies, Monoclonal
  • Integrins
  • Laminin
  • Receptors, Laminin