Inflammatory polyarthritis in mice transgenic for human T cell leukemia virus type I

Arthritis Rheum. 1993 Nov;36(11):1612-20. doi: 10.1002/art.1780361117.

Abstract

Objective: We have recently reported that arthropathy develops in high incidence among transgenic mice carrying the pX region of human T cell leukemia virus type I (HTLV-I). In the present study, the histopathologic features of the joints in these mice were examined in order to compare the animal disease with rheumatoid arthritis (RA) in humans.

Methods: Paraffin sections of limbs (right and left fingers, wrists, elbows, shoulders, toes, knees, and ankles) were stained with hematoxylin and eosin, periodic acid-Schiff, azan-Mallory, or phosphotungstic acid hematoxylin, and examined by light microscopy.

Results: Abnormalities of the limbs began to occur as early as 3 weeks of age, and the incidence gradually increased until the mice were 12 months old. The incidence of arthropathy was 22% (48 of 217) at 3 months of age and 28% (18 of 64) at 6 months. The severity of the histopathologic changes in the joints of the transgenic mice ranged from grade I to grade IV.

Conclusion: The major histopathologic features in the joints of HTLV-I transgenic mice are similar to those in humans with RA. Thus, these mice may represent a useful model for the study of the disease in humans.

MeSH terms

  • Animals
  • Arthritis / etiology*
  • Arthritis / pathology
  • Blood Vessels / pathology
  • Human T-lymphotropic virus 1 / genetics*
  • Joints / blood supply
  • Joints / microbiology
  • Joints / pathology
  • Mice
  • Mice, Inbred C3H
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Synovial Membrane / pathology