Treatment of experimental visceral leishmaniasis in a T-cell-deficient host: response to amphotericin B and pentamidine

Antimicrob Agents Chemother. 1993 Jul;37(7):1504-5. doi: 10.1128/AAC.37.7.1504.

Abstract

In experimental visceral leishmaniasis, euthymic but not athymic (nude) BALB/c mice respond to conventional treatment with pentavalent antimony, indicating that the in vivo efficacy of antimony is T cell dependent. This finding correlates with frequent antimony treatment failures for T-cell-deficient patients with visceral leishmaniasis. To determine whether the in vivo efficacies of alternative antileishmanial agents also require T cells, Leishmania donovani-infected euthymic and nude BALB/c mice were treated with pentamidine or amphotericin B. Pentamidine induced leishmanistatic activity in euthymic mice but had little effect in nude mice. In contrast, amphotericin B exerted potent leishmanicidal activities in both euthymic and nude animals. These results suggest that amphotericin B may be of particular use for T-cell-deficient patients with visceral leishmaniasis.

Publication types

  • Comparative Study
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amphotericin B / pharmacology*
  • Animals
  • Disease Models, Animal
  • Female
  • Immunocompromised Host*
  • Immunosuppression Therapy
  • Leishmania donovani*
  • Leishmaniasis, Visceral / drug therapy*
  • Leishmaniasis, Visceral / immunology
  • Mice
  • Mice, Inbred BALB C
  • Mice, Nude
  • Pentamidine / pharmacology*
  • T-Lymphocytes / physiology*

Substances

  • Pentamidine
  • Amphotericin B