Abstract
Adhesion to, and interaction with, airway endothelium, interstitial matrix and epithelium during migration to the allergen-challenged airways of allergic rhinitis and asthma patients may account for the observed functional upregulation (priming) of airway eosinophils compared with corresponding blood eosinophils.
MeSH terms
-
Allergens / immunology
-
Amino Acid Sequence
-
Asthma / immunology*
-
Bronchoalveolar Lavage Fluid / cytology
-
Cell Adhesion / physiology
-
Cell Adhesion Molecules / blood*
-
Cell Survival / physiology
-
Eosinophils / immunology*
-
Humans
-
Hypersensitivity / immunology*
-
Molecular Sequence Data
-
Rhinitis, Allergic, Seasonal / immunology
Substances
-
Allergens
-
Cell Adhesion Molecules