Modulation of interferon-gamma-induced HLA-DR expression on the human keratinocyte cell line SCC-13 by ultraviolet radiation

Photochem Photobiol. 1993 Feb;57(2):285-90. doi: 10.1111/j.1751-1097.1993.tb02288.x.

Abstract

Cell surface expression of major histocompatibility determinants on epidermal keratinocytes is a characteristic feature of a number of inflammatory dermatoses and in all likelihood is caused by diffusion of human leukocyte antigen (HLA)-DR-inducing cytokines from cells present in the dermal mononuclear cell infiltrate. Many of these same disorders respond to ultraviolet (UV) radiation phototherapy. Using the human SCC-13 keratinocyte cell line as a model, UV radiation was found to inhibit interferon-gamma-induced HLA-DR expression. Inhibition correlated closely with decreased steady-state levels of HLA-DR mRNA. These findings provide evidence that the therapeutic effect of UV radiation phototherapy may be mediated by its capacity to down-regulate cytokine-induced keratinocyte HLA-DR expression.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Cell Line
  • Dinoprostone / pharmacology
  • Dose-Response Relationship, Radiation
  • Genes, MHC Class II / radiation effects
  • HLA-DR Antigens / biosynthesis
  • HLA-DR Antigens / genetics*
  • Humans
  • Indomethacin / pharmacology
  • Interferon-gamma / pharmacology*
  • Keratinocytes / immunology
  • Keratinocytes / radiation effects*
  • RNA, Messenger / metabolism
  • Recombinant Proteins
  • Ultraviolet Rays*

Substances

  • HLA-DR Antigens
  • RNA, Messenger
  • Recombinant Proteins
  • Interferon-gamma
  • Dinoprostone
  • Indomethacin