Retinoic acid decreases attachment of JAR choriocarcinoma spheroids to a human endometrial cell monolayer in vitro

Placenta. 1993 Jan-Feb;14(1):13-24. doi: 10.1016/s0143-4004(05)80245-0.

Abstract

The ability of retinoic acid (RA) to modulate attachment of JAR choriocarcinoma multicellular spheroids to monolayers of a human uterine epithelial cell line (RL95-2) was examined using a centrifugal force-based adhesion assay. Exposure of choriocarcinoma spheroids to RA (10(-7) to 10(-5) M) over a 3-day culture period resulted in a dose-dependent decrease of attachment. Significant decreases in attachment were detected after 30 min (75 per cent versus 25 per cent) and 1 h (92 per cent versus 26 per cent) of confrontation-culture between choriocarcinoma and uterine cells for control versus 10(-5) M RA; by 5 h 100 per cent spheroid attachment was detected in all treatment groups. RA had no effect on cell proliferation in JAR spheroids, but 10(-5) M RA treatment induced a fivefold increase in secretion of human chorionic gonadotrophin (hCG), a known marker of conversion of cytotrophoblast to syncytiotrophoblast-like cells. These findings demonstrate that RA modulates cellular attachment and differentiation in choriocarcinoma spheroids in vitro.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Adhesion / drug effects
  • Cell Differentiation / drug effects
  • Cell Division / drug effects
  • Choriocarcinoma / metabolism
  • Choriocarcinoma / pathology*
  • Chorionic Gonadotropin / metabolism
  • Endometrium / cytology
  • Endometrium / drug effects*
  • Female
  • Humans
  • Tretinoin / pharmacology*
  • Tumor Cells, Cultured

Substances

  • Chorionic Gonadotropin
  • Tretinoin