Downregulation of HLA class I expression by c-myc in human melanoma is independent of enhancer A

Nucleic Acids Res. 1993 Mar 11;21(5):1179-85. doi: 10.1093/nar/21.5.1179.

Abstract

High constitutive expression of the c-myc oncogene in human melanoma leads to downregulation of expression of HLA Class I genes. The genes at the HLA-B locus are preferentially affected. To investigate the mechanism of downregulation, the activity of the main HLA Class I enhancer, enhancer A-region I, was compared in a panel of c-myc transfectants with increasing myc expression. Gel retardation experiments demonstrated in all tested cell lines binding of the transcription factors KBF1 and NF-kappa B to the enhancer. However, no correlation between the levels of HLA Class I expression and binding to the enhancer could be established. Strikingly, the cell line with the highest c-myc expression showed more binding of KBF1 and NF-kappa B than the parental cell line. By using CAT reporter plasmids in transient transfection assays we investigated the in vivo function of enhancer A-region I in the c-myc transfectant panel. Again, c-myc expression had no effect at all on the activity of enhancer A. This study shows that HLA Class I expression is regulated by the c-myc oncogene at the level of transcription, but that the main HLA Class I enhancer is not involved in this process.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • DNA Probes
  • Down-Regulation
  • Enhancer Elements, Genetic*
  • Gene Expression Regulation, Neoplastic*
  • Genes, MHC Class I*
  • Humans
  • L Cells
  • Melanoma / genetics*
  • Melanoma / immunology
  • Mice
  • Molecular Sequence Data
  • Protein Binding
  • Proto-Oncogene Proteins c-myc / genetics*
  • Proto-Oncogene Proteins c-myc / metabolism
  • Transcription, Genetic
  • Tumor Cells, Cultured

Substances

  • DNA Probes
  • Proto-Oncogene Proteins c-myc