Permeability of the blood-brain barrier to melanocortins

Peptides. 1995;16(6):1157-61. doi: 10.1016/0196-9781(95)00043-j.

Abstract

Adrenocorticotropin (ACTH)-related compounds, termed melanocortins, produce a large number of effects on the central nervous system (CNS) after their peripheral administration. Some of the CNS effects of ACTH are mediated through the release of glucocorticoids from the adrenal gland, but there are fragments and analogues of ACTH that do not act on the adrenals. This raises the possibility that some blood-borne melanocortins may be acting directly on the brain, which would necessitate their crossing the blood-brain barrier (BBB). We review here the literature showing that melanocortins can affect the BBB in several ways, including an alteration of the permeability of the BBB to other substances.

Publication types

  • Research Support, U.S. Gov't, Non-P.H.S.
  • Review

MeSH terms

  • Adrenocorticotropic Hormone / analogs & derivatives
  • Adrenocorticotropic Hormone / blood
  • Adrenocorticotropic Hormone / metabolism*
  • Amino Acid Sequence
  • Animals
  • Biological Transport, Active
  • Blood-Brain Barrier / physiology*
  • Humans
  • Melanocyte-Stimulating Hormones / blood
  • Melanocyte-Stimulating Hormones / chemistry
  • Melanocyte-Stimulating Hormones / metabolism
  • Molecular Sequence Data
  • Peptide Fragments / blood
  • Peptide Fragments / chemistry
  • Peptide Fragments / metabolism
  • Permeability

Substances

  • Peptide Fragments
  • Adrenocorticotropic Hormone
  • Melanocyte-Stimulating Hormones