Protection against osmotic stress by cGMP-mediated myosin phosphorylation

Science. 1996 Jan 12;271(5246):207-9. doi: 10.1126/science.271.5246.207.

Abstract

Conventional myosin functions universally as a generator of motive force in eukaryotic cells. Analysis of mutants of the microorganism Dictyostelium discoideum revealed that myosin also provides resistance against high external osmolarities. An osmo-induced increase of intracellular guanosine 3',5'-monophosphate was shown to mediate phosphorylation of three threonine residues on the myosin tail, which caused a relocalization of myosin required to resist osmotic stress. This redistribution of myosin allowed cells to adopt a spherical shape and may provide physical strength to withstand extensive cell shrinkage in high osmolarities.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actin Cytoskeleton / chemistry
  • Actins / analysis
  • Animals
  • Cyclic GMP / analogs & derivatives
  • Cyclic GMP / metabolism*
  • Cyclic GMP / pharmacology
  • Cytoplasm / chemistry
  • Dictyostelium / genetics
  • Dictyostelium / physiology*
  • Dictyostelium / ultrastructure
  • Glucose / pharmacology
  • Guanylate Cyclase / metabolism
  • Myosins / analysis
  • Myosins / metabolism*
  • Osmotic Pressure
  • Phosphorylation
  • Pseudopodia / chemistry
  • Pseudopodia / ultrastructure
  • Threonine / metabolism
  • Water-Electrolyte Balance

Substances

  • Actins
  • Threonine
  • 8-bromocyclic GMP
  • Myosins
  • Guanylate Cyclase
  • Cyclic GMP
  • Glucose