Bispecific monoclonal antibodies for intravenous treatment of carcinoma patients: immunobiologic aspects

J Hematother. 1995 Oct;4(5):409-14. doi: 10.1089/scd.1.1995.4.409.

Abstract

Immunobiologic parameters measured during a phase I trial of intravenously (i.v.) administered bispecific monoclonal antibodies (BsmAb) in renal cell carcinoma (RCC) patients are described. The BsmAb used, BIS-1, is reactive with a pancarcinoma-associated 38 kDa transmembrane glycoprotein, EGP-2, as well with the CD3 complex. Patients received during a 2 h i.v. infusion F(ab')2 fragments of BIS-1 at doses of 1, 3, or 5 micrograms/kg body weight during concomitantly applied subcutaneous (s.c.) IL-2 treatment. Acute but transient BIS-1 F(ab')2-related toxicity was observed at the 3 and 5 micrograms/kg dose level, and the maximum tolerated dose (MTD) was set at 5 micrograms/kg. A dose-dependent binding of BIS-1 F(ab')2 to circulating T lymphocytes was found. The in vivo occupancy of CD3 molecules on T lymphocytes was highest at teh end of the infusion period and then rapidly decreased, as shown by flow cytometry. A much slower decrease of BIS-1 F(ab')2 binding was observed in vitro, suggesting migration of BIS-1 F(ab')2-loaded T lymphocytes from the circulation. A strong but transitory leukopenia was observed, in which LFA-1 alpha bright, CD3/CD8 double positive T cells left the circulation preferentially. This phenomenon was most likely induced by elevated TNF-alpha and IFN-gamma plasma levels, which were at a maximum shortly after the end of the infusion. Isolated peripheral blood mononuclear cells obtained from patients directly after treatment with BIS-1 F(ab')2 at the 3 and 5 micrograms/kg dose level showed increased EGP-2-directed antitumor activity.

Publication types

  • Clinical Trial
  • Clinical Trial, Phase I
  • Controlled Clinical Trial

MeSH terms

  • Antibodies, Bispecific / administration & dosage
  • Antibodies, Bispecific / adverse effects
  • Antibodies, Bispecific / immunology
  • Antibodies, Bispecific / therapeutic use*
  • Antibodies, Monoclonal / administration & dosage
  • Antibodies, Monoclonal / adverse effects
  • Antibodies, Monoclonal / immunology
  • Antibodies, Monoclonal / therapeutic use*
  • Antibodies, Neoplasm / administration & dosage
  • Antibodies, Neoplasm / adverse effects
  • Antibodies, Neoplasm / immunology
  • Antibodies, Neoplasm / therapeutic use*
  • Antigens, Neoplasm / immunology*
  • Carcinoma, Renal Cell / immunology
  • Carcinoma, Renal Cell / therapy*
  • Cell Adhesion Molecules / immunology*
  • Combined Modality Therapy
  • Cytotoxicity, Immunologic
  • Dose-Response Relationship, Immunologic
  • Epithelial Cell Adhesion Molecule
  • Humans
  • Immunization, Passive / adverse effects
  • Immunoglobulin Fab Fragments / administration & dosage
  • Immunoglobulin Fab Fragments / adverse effects
  • Immunoglobulin Fab Fragments / immunology
  • Immunoglobulin Fab Fragments / therapeutic use*
  • Immunologic Factors / administration & dosage
  • Immunologic Factors / therapeutic use
  • Injections, Intravenous
  • Injections, Subcutaneous
  • Interferon-gamma / blood
  • Interleukin-2 / administration & dosage
  • Interleukin-2 / adverse effects
  • Interleukin-2 / therapeutic use
  • Kidney Neoplasms / immunology
  • Kidney Neoplasms / therapy*
  • Leukopenia / chemically induced
  • Muromonab-CD3 / administration & dosage
  • Muromonab-CD3 / adverse effects
  • Muromonab-CD3 / immunology
  • Muromonab-CD3 / therapeutic use*
  • T-Lymphocyte Subsets / drug effects
  • T-Lymphocyte Subsets / immunology
  • Tumor Necrosis Factor-alpha / analysis

Substances

  • Antibodies, Bispecific
  • Antibodies, Monoclonal
  • Antibodies, Neoplasm
  • Antigens, Neoplasm
  • Cell Adhesion Molecules
  • Epithelial Cell Adhesion Molecule
  • Immunoglobulin Fab Fragments
  • Immunologic Factors
  • Interleukin-2
  • Muromonab-CD3
  • Tumor Necrosis Factor-alpha
  • Interferon-gamma