PLC gamma 1 Src homology domain induces mitogenesis in quiescent NIH 3T3 fibroblasts

Biochem Biophys Res Commun. 1996 May 6;222(1):186-93. doi: 10.1006/bbrc.1996.0719.

Abstract

Previously, we demonstrated that microinjection of phosphoinositide-specific phospholipase C gamma 1 (PLC gamma 1) and lipase-defective mutants of PLC gamma 1 induced G(0) growth arrested NIH 3T3 fibroblasts to enter S phase of the cell cycle. These experiments suggested that regions other than the catalytic domain of PLC gamma 1 may be responsible for inducing mitogenesis. To test other regions of PLC gamma 1 for DNA synthesis inducing activity, cDNA fragments encoding Src homology (SH) and pleckstrin homology (PH) domains were subcloned into the bacterial expression plasmid pGEX-2TK, and the GST fusion proteins were purified. The complete PLC gamma l SH domain peptide was found to induce DNA synthesis after microinjection into growth arrested fibroblasts. Peptides containing a single SH3 domain or two SH2 domains induced a partial response that was restored to full activity if they were co-injected. The PH domain peptide did not induce DNA synthesis. Thus, both SH3 and SH2 activity combine to give maximum DNA synthesis induction, demonstrating that non-catalytic structural domains of PLC gamma 1 have pronounced effects on mitogenic signaling pathways.

MeSH terms

  • 3T3 Cells
  • Animals
  • Blood Proteins
  • Isoenzymes / physiology*
  • Mice
  • Mitosis*
  • Phospholipase C gamma
  • Phosphoproteins*
  • Signal Transduction
  • Structure-Activity Relationship
  • Type C Phospholipases / physiology*
  • src Homology Domains

Substances

  • Blood Proteins
  • Isoenzymes
  • Phosphoproteins
  • platelet protein P47
  • Type C Phospholipases
  • Phospholipase C gamma