Processing and activation of CMH-1 by granzyme B

J Biol Chem. 1996 May 3;271(18):10816-20. doi: 10.1074/jbc.271.18.10816.

Abstract

Granzyme B plays an essential role in cytotoxic T lymphocyte (CTL)-mediated cell killing. Recent studies suggest that granzyme B may exert its effect by cleaving and activating CPP32, a member of the interleukin-1 beta-converting enzyme/Ced-3 family of cysteine proteases. We have examined the processing and activation of CMH-1, a close homologue of CPP32, by granzyme B in vitro. We have found that granzyme B specifically cleaves CMH-1 at Asp198-Ser199 between the p20 and p12 and activates the cysteine protease. Cleavage between p20 and the prosequence of CMH-1 at Asp23-Ala24 is autocatalytic and is not required for CMH-1 activity in vitro. The cleavage and activation of CMH-1 by granzyme B in vitro sugge st that, in addition to CPP32, CMH-1 may also play a role in CTL-mediated cell killing.

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Base Sequence
  • Caspase 7
  • Caspases*
  • Catalysis
  • Cell Death / immunology
  • Cell Line
  • Cysteine Endopeptidases / genetics
  • Cysteine Endopeptidases / metabolism*
  • DNA, Complementary
  • Enzyme Activation
  • Granzymes
  • Hydrolysis
  • Molecular Sequence Data
  • Protein Processing, Post-Translational*
  • Serine Endopeptidases / metabolism*
  • Substrate Specificity
  • T-Lymphocytes, Cytotoxic / immunology

Substances

  • DNA, Complementary
  • Granzymes
  • Serine Endopeptidases
  • Caspase 7
  • Caspases
  • Cysteine Endopeptidases