LPS does not directly induce STAT activity in mouse macrophages

Cell Immunol. 1996 May 25;170(1):20-4. doi: 10.1006/cimm.1996.0129.

Abstract

Induction of gene expression in cytokine-treated cells involves the protein tyrosine kinase-dependent activation of members of the STAT family of transcription factors. To determine if lipopolysaccharide (LPS) might activate one or more STAT factors, nuclear extracts from LPS-treated RAW264.7 macrophages were assayed for STAT-like DNA binding activity using oligonucleotides recognized by different members of this protein family. Within 30 min a single LPS-inducible DNA-protein complex was detected using three separate oligonucleotides. This activity was not reactive with anti-STAT antibodies and was subsequently identified as composed of the NF kappa B components NF kappa B1 and Rel-A. Thus, LPS does not directly stimulate STAT factors with known sequence-specific DNA binding activity.

MeSH terms

  • Animals
  • Antibodies / chemistry
  • Base Sequence
  • Cell Line
  • DNA-Binding Proteins / biosynthesis*
  • DNA-Binding Proteins / drug effects
  • DNA-Binding Proteins / immunology
  • Lipopolysaccharides / pharmacology*
  • Macrophages / drug effects
  • Macrophages / metabolism*
  • Mice
  • Molecular Sequence Data
  • NF-kappa B / metabolism
  • Protein Binding / immunology
  • STAT1 Transcription Factor
  • Signal Transduction / drug effects*
  • Trans-Activators / biosynthesis*
  • Trans-Activators / drug effects
  • Trans-Activators / immunology

Substances

  • Antibodies
  • DNA-Binding Proteins
  • Lipopolysaccharides
  • NF-kappa B
  • STAT1 Transcription Factor
  • Stat1 protein, mouse
  • Trans-Activators