Pioglitazone hydrochloride stimulates insulin secretion in HIT-T 15 cells by inducing Ca2+ influx

J Endocrinol. 1996 Jul;150(1):107-11. doi: 10.1677/joe.0.1500107.

Abstract

Pioglitazone hydrochloride (AD-4833), one of the thiazolidinedione analogs, is a new anti-diabetic agent which improves peripheral insulin resistance in diabetic patients. We determined the direct effect of AD-4833 on insulin secretion in HIT-T 15 cells. The effects of AD-4833 (10(-7) M to 10(-5) M) on insulin secretion were examined in 3 and 7 mM glucose-containing F-12 K media. The addition of 10(-5) M AD-4833 significantly increased insulin secretion in both media, but its stimulatory effect was more potent in the medium containing 7 mM glucose. The addition of 10(-5) M AD-4833 caused an immediate, significant increase in cytosolic free Ca2+ concentration ([Ca2+]i). Nifedipine at all concentrations from 10 to 1000 nM significantly attenuated insulin secretion by 10(-5) M AD-4833. In addition, 10(-5) M AD-4833 failed to stimulate insulin secretion in the CA(2+)-free Kreb's-Ringer bicarbonate buffer. These data indicated that AD-4833 stimulates in vitro insulin secretion in HIT-T 15 cells, perhaps by inducing Ca2+ influx.

MeSH terms

  • Animals
  • Calcium / metabolism*
  • Calcium Channel Blockers / pharmacology
  • Cell Line
  • Cricetinae
  • Glucose / metabolism
  • Hypoglycemic Agents*
  • Insulin / metabolism*
  • Insulin Secretion
  • Islets of Langerhans / drug effects
  • Islets of Langerhans / metabolism*
  • Nifedipine / pharmacology
  • Pioglitazone
  • Stimulation, Chemical
  • Thiazoles / pharmacology*
  • Thiazolidinediones*

Substances

  • Calcium Channel Blockers
  • Hypoglycemic Agents
  • Insulin
  • Thiazoles
  • Thiazolidinediones
  • Nifedipine
  • Glucose
  • Calcium
  • Pioglitazone