[Subarachnoid administration of lidocaine reduces delayed neuronal damage due to forebrain ischemia in rats]

Masui. 1996 Apr;45(4):421-7.
[Article in Japanese]

Abstract

We examined the effect of subarachnoid administration of lidocaine on delayed neuronal damage due to forebrain ischemia in rats. Sixteen rats were divided at random into two groups: lidocaine group rats (n = 8) and control group rats (n = 8) were subarachnoidally administrated lidocaine 5mg.kg-1 and normal saline, respectively, prior to ischemia. Forebrain ischemia was induced by bilateral carotid artery occlusion combined with systemic hypotension for 10 min. Cerebral perfusion-fixation was performed 7 days after ischemia, subsequent to which the brains were sectioned coronally and stained with celestine blue/acid fuchsin. In the neocortex and the hippocampal CA4 region, the lidocaine group showed less ischemic neuronal injuries than the control group. However, in the hippocampal CA1/CA3 regions and the caudoputamen, neuronal injuries in the lidocaine group were not significantly different from those in the control group. We conclude that the pre-ischemic subarachnoid administration of lidocaine reduces the delayed neuronal damage in neocortex and hippocampal CA4 region in the rat with forebrain ischemia.

Publication types

  • English Abstract

MeSH terms

  • Anesthetics, Local / administration & dosage*
  • Animals
  • Brain Ischemia / drug therapy*
  • Brain Ischemia / pathology
  • Cell Death
  • Lidocaine / administration & dosage*
  • Male
  • Neurons / pathology*
  • Prosencephalon / pathology
  • Rats
  • Rats, Sprague-Dawley
  • Subarachnoid Space

Substances

  • Anesthetics, Local
  • Lidocaine